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MRCEM Part C · OSCE Preparation

OSCE Station Bank 6

10 new structured stations — respiratory emergencies, neurology, critical care procedures, difficult communication scenarios and advanced ECG interpretation.

0/ 10 completed
10Stations
5Domain types
8Min / station
0/10 completed
📋 History Taking
📋
Dyspnoea History — Acute Wheeze
History · 8 minStation 1 of 10
8:00
Station type
History Taking
Time allowed
8 minutes
Pass mark
12 / 20
📄 Candidate Briefing
👁 Examiner Instructions
✅ Mark Scheme

📄 Candidate Instructions

You are the ED doctor. Miss Sarah Ahmed, 32 years old, presents with acute breathlessness and audible wheeze. She is a known asthmatic.

Triage obs: RR 28, SpO₂ 90% on air, HR 112, BP 126/80, PEFR 38% predicted. Speaking in short sentences.

Please take a focused history. You have 8 minutes.

💡 Key areas — SOCRATES, BTS Severity, Risk Factors ▼
  • SOCRATES for breathlessness: Onset (sudden vs gradual), duration, severity (PEFR, SpO₂, speech, accessory muscle use), associated wheeze, cough, sputum, chest tightness, chest pain, fever.
  • BTS severity classification: Moderate — PEFR 50–75%, no features of severe. Severe — PEFR 33–50%, RR ≥25, HR ≥110, inability to complete sentences. Life-threatening — PEFR <33%, SpO₂ <92%, silent chest, cyanosis, exhaustion, altered consciousness, hypotension. Near-fatal — raised PaCO₂.
  • Triggers: Allergens (pets, dust, pollen, mould), exercise, cold air, NSAID/aspirin, beta-blockers, infection (URTI preceding), stress, irritants (smoke, fumes).
  • Medication use: SABA frequency (≥3 uses/day = loss of control), ICS compliance (missed doses = high risk), LABA, montelukast, oral steroids. Previous ED visits for asthma.
  • Previous admissions and ITU: Any previous ITU admission = near-fatal asthma risk marker. Number of ED attendances in past year.
  • Background control: Daytime and nocturnal symptoms, activity limitation, personal best PEFR, last review with GP/asthma nurse, written asthma action plan.
  • Allergies: Aspirin/NSAIDs — important both as trigger and drug safety. Beta-blockers contraindicated.

⚠️ Examiner / Role-player Instructions — Not for Candidate

You are Sarah Ahmed. You are visibly breathless and speak in short phrases — pause between sentences. The diagnosis is acute severe asthma triggered by cat allergen exposure. You have been forgetting your preventer inhaler for the past 2 weeks.

🎭 Patient Script ▼
  • Breathlessness: Came on over about an hour. Woke you from sleep. Audible wheeze at home. Tightness in chest. No fever, no productive cough.
  • Trigger: Spent the afternoon and evening at a friend's house who has two cats. "I know I'm allergic but it was a birthday party."
  • Medications: Clenil Modulite 200 mcg BD (ICS) — "I've been forgetting it... probably only taking it 2 or 3 times a week." Salbutamol MDI — used 4 times today, barely helped. No LABA.
  • Previous admissions: One ED attendance 18 months ago, treated and discharged. No ITU admission. No oral steroids in past 6 months.
  • Allergy: Aspirin — causes wheeze. No beta-blockers.
  • Control: Usually well controlled. Waking once a week with wheeze recently. No written action plan. No recent asthma review.
🔔 Examiner Cues ▼
  • If candidate hasn't asked about ICS compliance by 3 minutes: Patient volunteers — "I have a preventer but I keep forgetting to use it."
  • If candidate hasn't asked about previous ITU: Prompt — "Has she ever needed more than nebulisers in the past?"
  • At 7 minutes, ask candidate: "How would you classify the severity of this attack, and what is your immediate management?"
CriterionMarks
Breathlessness History
Onset, duration, progression and severity of breathlessness elicited2
Associated wheeze, cough, chest tightness, fever asked1
Severity Assessment
Correctly applies BTS classification — identifies acute severe (PEFR 38%, RR 28, HR 112, short sentences)2
Life-threatening features screened — SpO₂, silent chest, exhaustion, altered consciousness2
Triggers and Risk Factors
Cat allergen trigger identified1
Other triggers screened — aspirin/NSAID allergy specifically asked1
Medication History
ICS compliance assessed — non-compliance with Clenil identified2
SABA frequency today elicited — 4 uses (poor response)1
Background History
Previous ED attendances and ITU admission asked — key risk marker2
Oral steroid use in past year asked1
Written action plan and last asthma review asked1
Communication
Adapts communication to breathless patient — brief questions, allows pauses1
Total20
📋
Palpitations History — New AF
History · 8 minStation 2 of 10
8:00
Station type
History Taking
Time allowed
8 minutes
Pass mark
12 / 20
📄 Candidate Briefing
👁 Examiner Instructions
✅ Mark Scheme

📄 Candidate Instructions

You are the ED doctor. Mr David Chen, 58 years old, presents with a 2-hour history of palpitations and mild breathlessness.

Triage obs: HR 138 irregularly irregular, BP 112/74, SpO₂ 96%, RR 18. ECG shows atrial fibrillation.

Please take a focused history. You have 8 minutes.

💡 Key areas — AF History, CHA₂DS₂-VASc, HAS-BLED ▼
  • Palpitations: Onset (sudden vs gradual), duration, character (fast/slow, regular/irregular — "bag of worms"), associated symptoms (chest pain, dyspnoea, pre-syncope, syncope, diaphoresis).
  • CHA₂DS₂-VASc risk factors: Congestive heart failure, Hypertension, Age (≥75 = 2 points, 65–74 = 1 point), Diabetes mellitus, Stroke/TIA/thromboembolism (2 points), Vascular disease (prior MI, PAD, aortic plaque), female Sex category.
  • HAS-BLED bleeding risk: Hypertension (uncontrolled SBP >160), Abnormal renal/liver function, Stroke history, Bleeding history or predisposition, Labile INR, Elderly (>65), Drugs (antiplatelets, NSAIDs, alcohol).
  • Thyroid symptoms: Weight loss, heat intolerance, tremor, diarrhoea, anxiety, eye changes — thyrotoxicosis is a reversible cause of AF.
  • Other precipitants: Caffeine excess, alcohol (holiday heart syndrome), recreational drugs (cocaine, amphetamines), infection/sepsis, hypoxia, electrolyte disturbance, recent surgery.
  • Previous episodes: First episode or recurrent? If recurrent — paroxysmal AF? Any previous cardioversion? On any antiarrhythmics already?
  • Medications: Current medications — especially antihypertensives, rate/rhythm control agents, anticoagulants, NSAIDs. Allergy.

⚠️ Examiner / Role-player Instructions — Not for Candidate

You are Mr David Chen. You are anxious but comfortable at rest. This is your first episode of AF. CHA₂DS₂-VASc score is 2 (hypertension, diabetes). No previous anticoagulation. Admit to significant alcohol intake if asked directly.

🎭 Patient Script ▼
  • Onset: Sudden onset 2 hours ago while watching television. "It just started — my heart was racing and jumping all over the place." Mild breathlessness but no chest pain, no syncope.
  • CHA₂DS₂-VASc: Hypertension — yes, on amlodipine 5mg. Diabetes — yes, on metformin. Age 58 — not in high-risk age band. No previous stroke or TIA. No known heart failure or peripheral vascular disease.
  • Thyroid: No weight loss, no heat intolerance, no tremor. "My doctor checked my thyroid last year — it was fine."
  • Alcohol: If asked directly — "About 4 or 5 pints of beer most evenings... I know it's too much." Had a heavy drinking session last night.
  • Caffeine/drugs: Drinks 6 coffees a day. No recreational drugs. No smoking.
  • Previous episodes: "No, never felt like this before." No previous AF diagnosis. No antiarrhythmics.
  • Medications: Amlodipine 5mg, Metformin 1g BD. No anticoagulants. No allergies.
🔔 Examiner Cues ▼
  • If candidate hasn't asked about alcohol by 4 minutes: Patient volunteers — "I did have quite a few drinks last night, actually."
  • If candidate hasn't asked about thyroid: Prompt — "Are there any underlying conditions that commonly cause this arrhythmia you'd want to exclude?"
  • At 7 minutes, ask: "What is the CHA₂DS₂-VASc score and what are your immediate management priorities?"
CriterionMarks
Palpitations History
Onset, duration and character of palpitations fully elicited2
Associated symptoms — chest pain, syncope, dyspnoea asked1
CHA₂DS₂-VASc Assessment
Hypertension and diabetes identified as risk factors2
Stroke/TIA history, heart failure, vascular disease screened2
Age risk stratification applied — correctly scores 2 (HTN + DM, age <65)1
Precipitants
Alcohol intake asked — significant excess identified2
Thyroid symptoms systematically screened2
Caffeine, drugs, infection, recent surgery asked1
Previous Episodes and Medications
First episode vs recurrent confirmed — first episode established1
Current medications and anticoagulation status clarified1
Communication
Systematic, structured history. Checks understanding and addresses concern2
Total20
🩺 Clinical Examination
🩺
Neurological Examination — Lower Limb
Examination · 8 minStation 3 of 10
8:00
Station type
Examination
Time allowed
8 minutes
Pass mark
12 / 20
📄 Candidate Briefing
👁 Examiner Instructions
✅ Mark Scheme

📄 Candidate Instructions

A 55-year-old man presents with right leg weakness and an abnormal gait after a road traffic accident 6 hours ago. CT head and cervical spine have been performed.

Please perform a focused lower limb neurological examination. The examiner will provide findings. Present your findings and localise the lesion.

🔑 Structured Lower Limb Neuro Exam ▼
  • Inspection: Muscle wasting (compare sides), fasciculations, deformity, skin changes, pressure sores, footwear.
  • Tone: Passive flexion/extension at hip, knee, ankle. Clonus — sustained ankle clonus ≥3 beats = UMN. Spasticity vs flaccidity.
  • Power (MRC 0–5): Hip flexion L1/2 (iliopsoas), hip extension L4/5 (glutei), knee extension L3/4 (quadriceps), knee flexion L5/S1 (hamstrings), ankle dorsiflexion L4/5 (tibialis anterior), ankle plantarflexion S1/2 (gastrocnemius), big toe extension L5 (EHL).
  • Reflexes: Knee L3/4 (patellar), Ankle S1/2 (Achilles). Plantar response — Babinski: flexor (normal) vs extensor (UMN). Reinforcement (Jendrassik manoeuvre) if absent.
  • Coordination: Heel-shin test (cerebellar — ipsilateral). Foot-tapping.
  • Sensation: Light touch and pin-prick dermatomal. Vibration (128Hz tuning fork at big toe then malleolus then knee). Joint position sense (JPS) at big toe.
  • Gait: Normal, antalgic, steppage (foot drop — L4/5), circumduction (spastic — UMN), ataxic (cerebellar), parkinsonian, Trendelenburg.
  • UMN vs LMN pattern: UMN — increased tone, hyperreflexia, extensor plantar, ± clonus, minimal wasting. LMN — flaccid, hyporeflexia/areflexia, flexor plantar, fasciculations, significant wasting.

⚠️ Examiner Instructions — Not for Candidate

Findings represent a right-sided UMN lesion — consistent with contralateral hemisphere or ipsilateral cord pathology. Feed findings progressively as candidate examines each component.

📋 Findings to Feed ▼
  • Inspection: No wasting. No fasciculations. Mild foot drop posture at rest right.
  • Tone: Increased tone right leg — clasp-knife spasticity at knee and ankle. Clonus present right ankle (5 beats). Normal tone left.
  • Power: Right — hip flexion 4/5, knee extension 4/5, ankle dorsiflexion 2/5, plantarflexion 3/5. Left — 5/5 throughout.
  • Reflexes: Right knee — brisk (+++) with cross-adductor response. Right ankle — brisk (+++). Left — normal (++). Plantar right — extensor (Babinski positive). Plantar left — flexor (normal).
  • Coordination: Heel-shin — right leg clumsy and overshoots. Left normal.
  • Sensation: Reduced light touch and pin-prick right leg below knee. Vibration absent right big toe, present at right knee. JPS impaired right big toe.
  • Gait: Right leg circumduction — classic spastic hemiplegic gait.
CriterionMarks
Inspection
Inspects both limbs — correctly notes no wasting, no fasciculations1
Tone
Tone assessed correctly both sides — spasticity and clonus identified right2
Power
All major muscle groups tested with correct MRC grading both sides2
Correct root values stated for tested muscles (at least 3 correct)1
Reflexes
Knee and ankle reflexes elicited bilaterally — hyperreflexia right documented2
Plantar responses tested — Babinski positive right, flexor left2
Sensation and Coordination
Light touch and pin-prick tested dermatomally — sensory loss right documented1
Vibration and JPS tested — impaired right1
Heel-shin coordination tested — right ataxia noted1
Gait and Diagnosis
Gait assessed — circumduction (spastic gait) described1
Correctly identifies UMN pattern and localises lesion — contralateral hemisphere or ipsilateral cord2
States need for urgent MRI spine/CT/MRI brain as appropriate1
Total20
🩺
Cranial Nerve Examination
Examination · 8 minStation 4 of 10
8:00
Station type
Examination
Time allowed
8 minutes
Pass mark
12 / 20
📄 Candidate Briefing
👁 Examiner Instructions
✅ Mark Scheme

📄 Candidate Instructions

A 45-year-old woman presents to ED with a 2-day history of right facial weakness and horizontal diplopia. She is otherwise systemically well. GCS 15.

Please perform a systematic cranial nerve examination. The examiner will provide findings. State your diagnosis and management plan.

🔑 Cranial Nerve Examination — CN I to XII ▼
  • CN I (Olfactory): Test each nostril separately — "Can you smell this?" (coffee, peppermint). Usually not tested unless specifically indicated.
  • CN II (Optic): Visual acuity (Snellen), visual fields (confrontation), colour vision (Ishihara), pupil reactions (direct + consensual + RAPD), fundoscopy (disc pallor = optic atrophy, papilloedema).
  • CN III, IV, VI (Oculomotor, Trochlear, Abducens): Inspect for ptosis, proptosis. Pupil size and reactivity. Extra-ocular movements — H-pattern. Ask about diplopia (worse in which direction). CN III = most movements + pupil (complete CN III = ptosis + down-and-out + fixed dilated pupil). CN IV = intorsion deficit (SOV). CN VI = failure to abduct (lateral rectus).
  • CN V (Trigeminal): Facial sensation — light touch and pin-prick V1 (forehead), V2 (cheek), V3 (jaw) bilaterally. Corneal reflex (afferent V, efferent VII). Jaw opening against resistance (pterygoids — deviation to weak side). Jaw jerk.
  • CN VII (Facial): Inspect at rest — facial asymmetry, nasolabial fold flattening, drooping. Raise eyebrows, close eyes tightly (can examiner open them?), puff cheeks, show teeth, smile. UMN lesion — spares forehead (bilateral cortical representation). LMN lesion — forehead involved (Bell's phenomenon).
  • CN VIII (Vestibulocochlear): Hearing — finger rub each ear. Rinne (512Hz tuning fork — AC>BC = normal or SNHL; BC>AC = conductive). Weber (lateralises to good ear in SNHL, affected ear in conductive).
  • CN IX, X (Glossopharyngeal, Vagus): Voice — hoarseness. Swallowing. Palate elevation ("say Aah") — deviation to unaffected side. Gag reflex (not routinely in conscious patients).
  • CN XI (Accessory): Sternocleidomastoid — turn head against resistance. Trapezius — shrug shoulders against resistance.
  • CN XII (Hypoglossal): Tongue — protrude, check for deviation (towards weak side), fasciculations (LMN), atrophy.

⚠️ Examiner Instructions — Not for Candidate

Findings represent right CN VII LMN palsy (Bell's palsy) and right CN VI palsy. If candidate does not differentiate UMN from LMN facial palsy, prompt: "Is the forehead spared or involved on the right side?"

📋 Findings to Feed ▼
  • CN I: Normal bilaterally.
  • CN II: VA 6/6 bilaterally. Fields full. Pupils equal, round, reactive 3mm → 2mm bilaterally. No RAPD. Fundoscopy normal.
  • CN III, IV: Normal — no ptosis, no pupil abnormality, full elevation/depression/adduction bilaterally.
  • CN VI: Right eye — failure to abduct fully on rightward gaze. Horizontal diplopia maximal on right lateral gaze. Left eye — full abduction.
  • CN V: Sensation intact V1–V3 bilaterally. Corneal reflex — present bilaterally (note: afferent CN V intact; efferent CN VII right — blink reduced right). Jaw — central, normal power.
  • CN VII: At rest — right nasolabial fold flat, slight drooping right corner of mouth. Eyebrow raise — right forehead wrinkles absent (forehead involved = LMN). Eye closure — right eye cannot close fully (Bell's phenomenon visible — eyeball rolls up). Puffing cheeks — right side deflates. Smile — markedly asymmetric. LMN pattern — forehead involved distinguishes from UMN.
  • CN VIII: Hearing intact bilaterally on finger rub. Rinne AC>BC bilaterally. Weber central.
  • CN IX–XII: All normal — voice clear, palate elevates symmetrically, tongue protrudes centrally, no fasciculations, SCM and trapezius normal.
CriterionMarks
CN II — Vision
Visual acuity, fields and pupil reactions tested — all normal documented2
CN III, IV, VI — Eye Movements
Extra-ocular movements tested in H-pattern — diplopia elicited on right lateral gaze2
Correctly identifies right CN VI palsy — failure to abduct right eye2
CN V — Trigeminal
Facial sensation tested V1–V3 bilaterally — correctly normal1
Corneal reflex tested — reduced right blink (CN VII efferent) with intact left1
CN VII — Facial
Forehead, eye closure and lower face all tested separately2
Correctly identifies LMN pattern — forehead involved (distinguishes from UMN/stroke)2
Remaining Nerves
CN VIII hearing tested — normal bilaterally1
CN IX/X — palate, voice. CN XI — SCM, trapezius. CN XII — tongue. All tested systematically.1
Diagnosis and Management
Correct diagnosis — right Bell's palsy (LMN CN VII) and right CN VI palsy2
Management — prednisolone within 72h, eye protection (lubricating drops, tape at night), ophthalmology if corneal exposure, MRI brain to exclude pontine lesion given CN VI also involved1
Total20
🔧 Practical Procedures
🔧
Central Line Insertion — Internal Jugular (Seldinger)
Procedure · 8 minStation 5 of 10
8:00
Station type
Procedure
Time allowed
8 minutes
Pass mark
12 / 20
📄 Candidate Briefing
👁 Examiner Instructions
✅ Mark Scheme

📄 Candidate Instructions

A 67-year-old man presents with septic shock secondary to pneumonia. He has no peripheral venous access after multiple attempts. He requires central venous access for vasopressors and ongoing resuscitation.

Obs: HR 122, BP 78/44, RR 28, Temp 38.9°C, SpO₂ 94% on 15L O₂.

Please talk through how you would insert a right internal jugular central venous catheter using the Seldinger technique. You have 8 minutes.

📝 CVC Insertion — Seldinger Technique ▼
  • Indications: No peripheral access, vasopressors, CVP monitoring, rapid large-volume resuscitation, TPN, haemodialysis, long-term IV access.
  • Site selection: RIJ preferred (straighter path to SVC, lower pneumothorax risk than subclavian, compressible if arterial puncture). Subclavian — higher PTX risk, lower infection rate. Femoral — high infection risk, avoid in coagulopathy/obese, no pneumothorax risk.
  • Contraindications/cautions: Coagulopathy (INR >1.5, platelets <50 — relative), infection overlying site, ipsilateral pneumothorax.
  • Preparation: Consent (if possible), position (Trendelenburg for RIJ/subclavian — increases vein size, reduces air embolism risk), turn head left, sterile technique (gown, gloves, drape, chlorhexidine prep).
  • USS guidance: Mandatory per NICE guidance. Identify IJ (compressible, thin-walled, lateral to carotid). Real-time USS guidance reduces complications.
  • Seldinger steps: 1. Needle into vein (confirm venous blood — non-pulsatile, dark). 2. Guidewire through needle. 3. Remove needle. 4. Skin nick with scalpel. 5. Dilator over wire. 6. Remove dilator. 7. CVC over wire. 8. Remove wire. 9. Aspirate and flush all lumens. 10. Secure and dress.
  • Confirmation: Blood flashback from all ports. CXR — tip at SVC/RA junction (carina level). Waveform if connected to transducer.
  • Complications: Immediate — arterial puncture (compress, remove, reassess), pneumothorax (CXR), air embolism (Trendelenburg, aspirate). Delayed — infection, thrombosis, catheter malposition.

⚠️ Examiner Instructions — Not for Candidate

Ask: "As you pass the guidewire you notice frequent ectopics on the monitor — what do you do?" (Expected: wire has entered right ventricle — withdraw wire to SVC level, ectopics should resolve). Also ask: "The post-procedure CXR shows a small right apical pneumothorax — what is your management?"

CriterionMarks
Preparation
Indications confirmed, consent obtained (or documented if unable), coagulopathy checked1
Site selection — RIJ chosen with justification. Trendelenburg positioning stated.2
Full aseptic technique — sterile gown, gloves, large drape, chlorhexidine prep2
USS guidance stated — real-time, per NICE guidance2
Seldinger Technique
Correct sequence — needle, wire, nick, dilator, CVC, wire removal, aspirate/flush3
Venous blood confirmed before wire insertion — non-pulsatile, dark1
Wire ectopics recognised — withdraws wire to SVC level2
Confirmation and Complications
CXR post-procedure — tip position at SVC/RA junction confirmed2
Complications named — arterial puncture, pneumothorax, air embolism, infection2
Pneumothorax management — if small and asymptomatic observe; if large or symptomatic drain1
Total20
🔧
Nasogastric Tube Insertion
Procedure · 8 minStation 6 of 10
8:00
Station type
Procedure
Time allowed
8 minutes
Pass mark
12 / 20
📄 Candidate Briefing
👁 Examiner Instructions
✅ Mark Scheme

📄 Candidate Instructions

A 72-year-old man with a reduced GCS of 9 (E2V3M4) following a large right MCA stroke is being managed in resus. He has no swallow reflex. The neurology team have requested nasogastric tube insertion for enteral feeding and medications.

Please talk through how you would insert an NGT, including relevant checks, the procedure, and how you confirm correct placement. You have 8 minutes.

📝 NGT Insertion — Key Points ▼
  • Indications: Enteral feeding (dysphagia, reduced consciousness), medication delivery, gastric decompression (ileus, bowel obstruction), gastric lavage (selected overdose).
  • Contraindications: Base of skull fracture (use orogastric route — risk of intracranial passage via cribriform plate), mid-face fracture, oesophageal stricture/varices/recent surgery, coagulopathy (relative).
  • Sizing: Fine-bore 8–10Fr for feeding. Wide-bore 14–16Fr for drainage/aspiration. Measure NEX length (Nose to Earlobe to Xiphisternum) to estimate insertion depth.
  • Technique: Position patient upright (30–45°) if possible. Lubricate tip with water-soluble gel. Insert along floor of nasal passage (horizontal, not upward). Advance gently — if resistance do not force. In conscious patient — ask to swallow sips of water as tube passes pharynx. In unconscious — flex neck if possible.
  • Confirmation of position (NPSA guidance): pH testing of gastric aspirate — pH ≤5.5 = safe to use. CXR if pH aspirate unobtainable or pH 5.5–6 (interpret carefully). Do NOT rely on auscultation alone ("whoosh test") — not reliable. Litmus paper alone not acceptable.
  • Securing: Secure to nose with appropriate tape. Mark tube at nostril. Document insertion depth. Re-check position before each feed/medication administration.
  • Complications: Pulmonary placement (most dangerous — feeds into lung), oesophageal placement, coiling in pharynx, nasal trauma/epistaxis, aspiration.

⚠️ Examiner Instructions — Not for Candidate

Ask: "The pH aspirate comes back as 6.2 — what do you do?" (Expected: cannot confirm safe — request CXR before using. Do not feed.) Also ask: "Why is the 'whoosh test' not acceptable as sole confirmation?"

CriterionMarks
Indications and Contraindications
Indication stated — dysphagia/reduced GCS requiring enteral nutrition1
Base of skull fracture as key contraindication — orogastric route alternative named2
Checks for mid-face trauma, oesophageal pathology1
Preparation and Technique
NEX measurement performed to estimate correct depth1
Correct positioning — 30–45° upright. Lubrication. Insertion along floor of nasal passage.2
Does not force if resistance. Swallowing manoeuvre mentioned if applicable.1
Confirmation of Position
pH testing of aspirate as primary method — pH ≤5.5 = safe2
pH 6.2 scenario — correctly states CXR required before use2
Correctly rejects "whoosh test" as unreliable sole confirmation — explains rationale2
Documentation and Complications
Documents position, depth at nostril, confirms before each feed/medication1
Pulmonary placement as key complication — recognition and management2
Nasal trauma, aspiration, coiling in pharynx mentioned1
Total20
💬 Communication
💬
Discharge Against Medical Advice
Communication · 8 minStation 7 of 10
8:00
Station type
Communication
Time allowed
8 minutes
Pass mark
12 / 20
📄 Candidate Briefing
👁 Examiner Instructions
✅ Mark Scheme

📄 Candidate Instructions

You are the ED registrar. Mr Kieran Walsh, 44 years old, was brought in by ambulance 90 minutes ago with a 3-hour history of central chest pain. His first troponin is pending (result due in 30 minutes). His ECG shows sinus rhythm with non-specific changes. He is currently pain-free.

The nurse has informed you that Mr Walsh is asking to self-discharge as he needs to get to work. He is alert and oriented.

Please speak with Mr Walsh. You have 8 minutes.

💡 Framework — DAMA Assessment ▼
  • Understand the reason: Explore why the patient wants to leave — work, family, anxiety, misunderstanding of situation. Address specific concern before anything else.
  • Assess mental capacity (MCA 2005): Does the patient understand the information? Retain it? Weigh it up? Communicate a decision? If all four — has capacity. A competent adult has the right to refuse treatment even if the decision appears unwise.
  • Explain the risks clearly: Troponin result is critical — result in 30 minutes. If elevated = heart attack requiring immediate intervention. If he leaves now and deteriorates at work, delay in treatment could be fatal. Risk of sudden cardiac death outside hospital.
  • Offer a compromise: Can he wait 30 minutes for the result? Can his employer be contacted? Can a family member come to support him? Is there anything you can do to make his stay more comfortable?
  • If he insists on leaving: Respect his decision if capacitous. Do not restrain or detain. Ask him to sign the DAMA form — but do not coerce. Document clearly. Provide written safety net — return immediately if pain returns, breathlessness, collapse, sweating, nausea.
  • Documentation: Record the conversation, capacity assessment, information given, risks explained, patient's understanding, decision and safety-netting in the notes.
  • Inform senior: Inform consultant/senior before patient leaves in high-risk situations.

⚠️ Examiner / Role-player Instructions — Not for Candidate

You are Mr Kieran Walsh. You are anxious but not angry. You are worried about your job — you manage a team and have a critical presentation at 2pm. You are not chest-pain-free but feel "fine now." You have no medical knowledge. If the candidate takes time to listen to your concerns and explains clearly what troponin is, you will agree to wait for the result. You are capacitous throughout.

🎭 Patient Script ▼
  • Opening: "Look, I feel fine now. I've been here for over an hour and no one's told me anything. I really need to go — I've got an important meeting at 2pm."
  • If asked about pain: "It's gone. I told you — I feel fine."
  • If asked about understanding: "I understand I had chest pain but it's gone now. What's the problem?"
  • When risks explained: Initially — "Surely it can't be that serious if I'm feeling OK?" Then softens if candidate explains clearly and empathically.
  • If candidate offers 30-minute compromise: "Alright... 30 minutes. But if it's nothing, I'm going. Can you ring me a taxi?"
  • Capacity: Fully capacitous. Understands, retains, weighs up, and communicates his decision clearly throughout.
🔔 Examiner Cues ▼
  • If candidate immediately reaches for DAMA form without assessing capacity: Stop — "Have you assessed his capacity?"
  • If candidate explains the troponin clearly and Mr Walsh agrees to stay: Mark as pass on communication domain.
  • At 7 minutes: "Mr Walsh asks — what happens if I don't sign the form?"
CriterionMarks
Engagement and Exploration
Introduces self, establishes rapport — does not immediately start with risks1
Explores and addresses the specific reason for leaving — work concern acknowledged2
Capacity Assessment
Mental capacity assessed — all four MCA criteria addressed (understand, retain, weigh up, communicate)3
Correctly identifies patient as capacitous — right to leave respected1
Risk Communication
Troponin explained in plain English — "a heart protein released if heart muscle is damaged"2
Risks of leaving explained clearly — cardiac arrest, missed MI, need for urgent intervention2
Compromise offered — wait 30 minutes for result, practical offer to help2
If Patient Still Insists on Leaving
Does not detain or coerce. DAMA form offered but not forced.1
Safety-netting provided — return if pain, breathlessness, sweating, collapse2
Documentation
States will document conversation, capacity assessment, risks explained and safety-netting in notes. Informs senior.1
Empathic, non-confrontational throughout. Does not use jargon.1
Total20
💬
Telephone Advice to GP — Chest Pain Referral
Communication · 8 minStation 8 of 10
8:00
Station type
Communication
Time allowed
8 minutes
Pass mark
12 / 20
📄 Candidate Briefing
👁 Examiner Instructions
✅ Mark Scheme

📄 Candidate Instructions

You are the ED registrar. You receive a phone call from Dr Priya Sharma, a GP, regarding a patient she saw in surgery 30 minutes ago. She is calling for advice about whether to send the patient to ED or manage in the community.

The patient is Mr James Norton, 62 years old. He presents with a 2-day history of central chest pain, worse on exertion, partially relieved by rest. New T-wave inversions in V4–V6 on the GP's ECG. He has not been sent in yet.

Please take the necessary history over the phone, risk-stratify the patient, give clear advice, and safety-net. You have 8 minutes.

💡 Key areas — HEART Score, Structured Advice ▼
  • Structured telephone history: Introduce yourself clearly. Confirm patient details. Take focused cardiac history — character of pain (exertional, radiation, sweating, nausea), duration, onset, progression. Risk factors — hypertension, diabetes, hypercholesterolaemia, smoking, family history, previous MI/PCI/CABG.
  • HEART score risk stratification: H = History (highly suspicious = 2, moderately suspicious = 1, slightly suspicious = 0). E = ECG (LBBB/ST changes = 2, non-specific/T-wave changes = 1, normal = 0). A = Age (≥65 = 2, 45–64 = 1, <45 = 0). R = Risk factors (≥3 or history of atherosclerosis = 2, 1–2 = 1, 0 = 0). T = Troponin (≥3x normal = 2, 1–3x = 1, normal = 0). Score 0–3 = low, 4–6 = moderate, ≥7 = high risk.
  • This scenario: Highly suspicious history (2) + T-wave inversions (1) + age 62 (1) + risk factors TBD + troponin unknown (0 assumed). Likely HEART 4–6 = intermediate risk minimum — needs ED assessment, serial troponins, cardiology review.
  • Clear advice: Admit immediately. Blue light ambulance if haemodynamically unstable or ongoing pain. Do not give aspirin until history clarified in ED. If patient stable and pain-free — urgent ED same day.
  • Safety-netting for GP: If patient deteriorates before ambulance arrives — call 999 immediately. Do not send patient alone by car. Give aspirin 300mg if pain is ongoing and no contraindication confirmed.
  • Documentation: Record the call, advice given, time, name of clinician called, and patient details. Follow up with formal admission.

⚠️ Examiner / Role-player Instructions — Not for Candidate

You are Dr Priya Sharma. You are a competent GP seeking specialist advice. Risk factors to disclose if asked: hypertension (controlled), type 2 diabetes, ex-smoker 10 years, no previous cardiac history. Patient is currently pain-free and stable in your surgery. You ask at the end: "Can I just give him an urgent cardiology OPD appointment instead of sending him to ED?"

🎭 GP Script ▼
  • Opening: "Hi, it's Dr Sharma from Westgate Surgery. I've got a 62-year-old male, Mr Norton, with 2 days of central exertional chest pain and new T-wave inversions. Just wanted to get your advice on whether to send him in."
  • If asked about character: "Central, tight, radiates to left arm, comes on walking up stairs, settles with rest. No pleuritic component, no positional change."
  • Risk factors: Hypertension on ramipril, T2DM on metformin, ex-smoker (stopped 10 years ago, 20 pack-year history). Cholesterol not checked recently. No previous MI, no stents.
  • Current status: "He's sitting in my waiting room, pain-free now, BP 148/88, HR 76 regular, SpO₂ 98%."
  • Challenge: At the end — "Can I send him to urgent cardiology OPD rather than ED?" (Expected: No — needs serial troponins and continuous monitoring. Cardiology OPD cannot provide this. Send to ED today.)
CriterionMarks
Communication Structure
Introduces self clearly — name and grade. Confirms patient details before proceeding.1
Structured telephone history — systematic, not disorganised1
History Taking
Cardiac character of pain elicited — exertional, radiation, associated symptoms2
Risk factors systematically assessed — HTN, DM, smoking, family history, previous cardiac events2
Current haemodynamic status and symptom status confirmed1
Risk Stratification
HEART score applied or equivalent systematic risk assessment — intermediate/high risk identified2
Correctly identifies need for serial troponins — cannot risk-stratify without this2
Advice and Safety-netting
Clear recommendation — send to ED today, not OPD. Justification given.2
Rejects cardiology OPD suggestion — explains serial troponin and monitoring requirement2
Safety-netting — if deteriorates call 999. Aspirin 300mg if ongoing pain, no contraindication.2
States will document the call — time, advice, clinician names1
Total20
📊 Data Interpretation
📊
ECG Interpretation — Complete Heart Block
Data · 8 minStation 9 of 10
8:00
Station type
Data Interpretation
Time allowed
8 minutes
Pass mark
12 / 20
📄 Candidate Briefing
👁 Examiner Instructions
✅ Mark Scheme

📄 Candidate Instructions

A 78-year-old man is brought in by ambulance after a syncopal episode at home. His wife reports he collapsed without warning and was unresponsive for approximately 2 minutes. He has regained consciousness but is drowsy and confused. He is on no cardiac medications.

Obs: HR 38, BP 78/46, RR 18, SpO₂ 96% on air, GCS 13 (E3V4M6).

The ECG shows: ventricular rate 38bpm, regular QRS complexes; atrial rate 76bpm, regular P waves; P waves and QRS complexes appear unrelated to each other; QRS broad at 160ms.

Please interpret this ECG and describe your immediate management. You have 8 minutes.

💡 Complete Heart Block — Diagnosis and Management ▼
  • ECG features of CHB (third-degree AV block): Regular P waves at their own rate (76bpm here). Regular QRS complexes at a slower escape rate (38bpm here). No relationship between P waves and QRS — complete AV dissociation. PR interval varies continuously. QRS broad if ventricular escape (below His-Purkinje), narrow if junctional escape (above bundle of His).
  • Differentiate from other AV blocks: First degree — prolonged PR >200ms, all P conducted. Second degree Mobitz I (Wenckebach) — progressive PR prolongation then dropped beat. Mobitz II — fixed PR, intermittently dropped QRS. 2:1 block — alternate P waves conducted. CHB — no P waves conducted at all.
  • Causes of CHB: Inferior MI (RCA → AV nodal artery — usually transient, narrow escape). Anterior MI (LAD → bundle branches — broad escape, poor prognosis). Drugs — beta-blockers, digoxin, calcium channel blockers, amiodarone. Lyme disease (young patient). Idiopathic fibrosis (Lev's/Lenègre's disease). Surgical complication (valve replacement, septal defect repair). Infiltrative — sarcoidosis, amyloidosis.
  • Immediate management: ABC. IV access. Continuous cardiac monitoring. 12-lead ECG. Urgent cardiology. Atropine 500mcg IV (may not work in infranodal block — broad QRS). Transcutaneous pacing if atropine fails or haemodynamically unstable. Temporary transvenous pacing (definitive bridge). Permanent pacemaker — definitive treatment.
  • Check medications: AV-nodal blocking drugs — withhold digoxin, rate-limiting calcium channel blockers, beta-blockers.

⚠️ Examiner Instructions — Not for Candidate

Ask: "You give atropine 500mcg IV — the rate does not improve. What do you do next?" (Expected: transcutaneous pacing — set rate 70–80bpm, increase current until capture, check for mechanical capture with pulse check). Also ask: "What additional investigation would change your immediate management priority?" (Expected: urgent 12-lead to look for inferior MI — STEMI with CHB = primary PCI immediately).

CriterionMarks
ECG Interpretation
Systematic approach — rate, rhythm, axis, P waves, QRS, ST/T stated1
Ventricular rate (38) and atrial rate (76) identified separately2
Complete AV dissociation identified — P waves and QRS unrelated2
Broad QRS escape rhythm — ventricular origin (below bundle of His)1
Correct diagnosis — complete (third-degree) AV block2
Causes
At least 3 causes named — inferior MI, drugs, Lyme disease, fibrosis, anterior MI2
Recognises inferior MI association — states urgent 12-lead + troponin to exclude STEMI1
Management
ABC, IV access, monitoring, urgent cardiology1
Atropine 500mcg IV — states may not work in infranodal (broad QRS) block2
Transcutaneous pacing — technique described correctly (rate 70–80, increase current until capture, check pulse)2
Temporary transvenous pacing and permanent pacemaker as definitive treatment1
Offending medications withheld — AV-nodal blocking drugs1
Total20
📊
ECG Interpretation — Ventricular Tachycardia
Data · 8 minStation 10 of 10
8:00
Station type
Data Interpretation
Time allowed
8 minutes
Pass mark
12 / 20
📄 Candidate Briefing
👁 Examiner Instructions
✅ Mark Scheme

📄 Candidate Instructions

A 65-year-old man with a history of previous MI presents with a 20-minute history of palpitations and presyncope. He is pale and diaphoretic. He is conscious and talking in full sentences.

Obs: HR 182, BP 88/54, RR 22, SpO₂ 96% on air.

The ECG shows: broad complex tachycardia, rate 182bpm, regular; QRS width 160ms; AV dissociation visible (P waves independent of QRS); fusion beats present in leads II and III; capture beats visible; positive concordance in chest leads V1–V6.

Please interpret this ECG, differentiate from SVT with aberrancy, and describe your management. You have 8 minutes.

💡 VT vs SVT with Aberrancy — Brugada Criteria ▼
  • Broad complex tachycardia — approach: Treat as VT until proven otherwise. Clinical context matters — previous MI, age, haemodynamic instability all increase VT likelihood.
  • Features confirming VT: AV dissociation (P waves independent of QRS — pathognomonic), fusion beats (partial ventricular activation by both sinus and ectopic pacemaker — pathognomonic), capture beats (normal narrow QRS interrupting broad complex — pathognomonic), QRS width >160ms, positive concordance in chest leads (all V1–V6 same direction — positive or negative), northwest axis (extreme axis -90° to ±180°).
  • Brugada criteria (4-step algorithm to diagnose VT): 1. Is there absence of RS complex in all chest leads? (VT if yes). 2. Is the RS interval >100ms in any chest lead? (VT if yes). 3. Is there AV dissociation? (VT if yes). 4. Are there morphological criteria for VT in V1–2 and V6? (VT if yes). If all 4 no — SVT with aberrancy.
  • DO NOT: Give verapamil to broad complex tachycardia — if VT → cardiovascular collapse. Do not use adenosine routinely in unstable patient.
  • Management — haemodynamically unstable VT: Synchronised DC cardioversion immediately (if pulse present) — 200J biphasic. Sedation if conscious (midazolam + fentanyl). If pulseless — treat as VF/pVT: unsynchronised defibrillation per ALS protocol.
  • Management — stable VT: Amiodarone 300mg IV over 20–60 minutes (loading dose), then 900mg over 24 hours. Correct electrolytes (K⁺ >4 mmol/L, Mg²⁺ 0.8–1.2 mmol/L). Cardiology involvement. If fails — synchronised cardioversion.
  • Post-conversion: Identify and treat cause (ischaemia, electrolytes, drugs). Cardiology review — ICD consideration if structural heart disease.

⚠️ Examiner Instructions — Not for Candidate

Ask: "A colleague suggests this could be SVT with aberrancy and recommends verapamil — what do you say?" (Expected: Do NOT give verapamil to broad complex tachycardia of uncertain origin. If VT — verapamil causes profound hypotension and VF. Treat as VT.) Also ask: "The patient suddenly becomes unresponsive with no palpable pulse — what is your next step?" (Expected: Unsynchronised defibrillation — pVT/VF, start ALS protocol.)

CriterionMarks
ECG Interpretation
Systematic approach — rate, rhythm, QRS width, axis documented1
Broad complex tachycardia at 182bpm correctly described1
AV dissociation identified — P waves independent of QRS2
Fusion beats and/or capture beats identified — pathognomonic of VT2
Positive concordance in V1–V6 identified as VT feature1
Correct diagnosis — ventricular tachycardia2
VT vs SVT Differentiation
Brugada criteria applied or equivalent systematic approach2
Correctly rejects verapamil — explains risk of cardiovascular collapse in VT2
Management
Haemodynamically unstable — synchronised DC cardioversion 200J stated (not amiodarone first)2
If pulseless — unsynchronised defibrillation, ALS protocol2
Electrolyte correction — K⁺ and Mg²⁺ targets stated. Cardiology involvement.1
Post-conversion — identify cause, ICD consideration in structural heart disease1
Does not use adenosine as first-line in unstable broad complex tachycardia1
Total20